What is the SVO2/SCVO2 status in distributive shock?

In distributive shock, particularly in cases such as septic shock, the status of mixed venous oxygen saturation (SVO2) or central venous oxygen saturation (SCVO2) can be variable. This occurs because, despite there being a significant drop in systemic vascular resistance and subsequent hypotension, the body may still be able to maintain or even increase the oxygen saturation due to several compensatory mechanisms.

Tissue perfusion may initially be preserved in the early stages of distributive shock, leading to normal or elevated levels of SVO2 or SCVO2. While there is hypotension and a reduction in effective circulatory volume, the increased cardiac output that can occur in response to the shock and the potential ability of tissues to extract less oxygen can contribute to this scenario. In certain conditions, the availability of oxygen may compensate for the reduced perfusion, leading to an increase in venous oxygen saturation despite the patient's overall compromised state.

Thus, it is accurate to state that in distributive shock, the SVO2 or SCVO2 may present as normal or even increased due to the underlying pathophysiology of the condition. This emphasizes the complexity of shock states and the importance of monitoring these parameters closely to guide appropriate therapeutic interventions.